Non-canonical NFκB activation promotes chemokine expression in podocytes
Por um escritor misterioso
Last updated 22 dezembro 2024
α-Actinin 4 Potentiates Nuclear Factor κ-Light-chain-enhancer of Activated B-cell (NF-κB) Activity in Podocytes Independent of Its Cytoplasmic Actin Binding Function - ScienceDirect
Non-canonical NFκB activation promotes chemokine expression in podocytes
NF-κB signaling in neoplastic transition from epithelial to mesenchymal phenotype, Cell Communication and Signaling
Non-canonical NF-κB signaling pathway. Simplified scheme of the
Cells, Free Full-Text
EGF-Receptor–Dependent TLR7 Signaling in Macrophages Promotes Glomerular Injury in Crescentic Glomerulonephritis - Laboratory Investigation
Frontiers Ferroptosis: an important player in the inflammatory response in diabetic nephropathy
TNFR2 interposes the proliferative and NF-κB-mediated inflammatory response by podocytes to TNF-α
Podocyte derived TNF‐α mediates monocyte differentiation and contributes to glomerular injury - Nishad - 2022 - The FASEB Journal - Wiley Online Library
The activation of canonical and non-canonical NF-κB signaling pathways
TWEAK/Fn14 and Non-Canonical NF-kappaB Signaling in Kidney Disease
NF-κB signaling pathways with receptors, inhibitors, targets, and other
IJMS, Free Full-Text
Overview of the canonical and non-canonical NF-κB signaling pathways.
Non-canonical NFκB activation promotes chemokine expression in podocytes
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